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Hantavirus inhibits apoptosis by preventing mitochondrial membrane potential loss through up-regulation of the pro-survival factor BCL-2

PLoS Pathog. 2020; 
Solà-Riera C, García M, Ljunggren HG, Klingstr?m J.
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Recombinant Proteins Plasmids expressing wild-type HTNV nucleocapsid protein (pCMV-Bios- HTNV-N), wild-type HTNV glycoproteins (pCMV-Bios-HTNV-G), and empty plasmid (pCMV-Bios-empty) were constructed by GenScript. Get A Quote

摘要

Hantaviruses, zoonotic RNA viruses belonging to the order Bunyavirales, cause two severe acute diseases in humans, hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS). Hantavirus-infected patients show strong cytotoxic lymphocyte responses and hyperinflammation; however, infected cells remain mostly intact. Hantaviruses were recently shown to inhibit apoptosis in infected cells. By inhibiting granzyme B- and TRAIL-mediated apoptosis, hantaviruses specifically and efficiently inhibit cytotoxic lymphocyte-mediated killing of infected cells. Hantaviruses also strongly inhibit apoptosis triggered intrinsically; i.e., initiated through intracellular activation pathways different from... More

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